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Aug 8, 2011 · This tutorial explains how to create barocdes in Microsoft Excel using the IDAutomation ...Duration: 1:51 Posted: Aug 8, 2011 a prolonged reaction time and bradykinesia with a prolonged time of execution, but he concedes that if bradykinesia is severe, it may result in akinesia This is apparently not the result of slowness in formulating the plan of movement; that is to say, there is no bradypraxia For a time, bradykinesia was attributed to the frequently associated rigidity, which could reasonably hamper all movements; but the incorrectness of this explanation became apparent when it was discovered that an appropriately placed stereotactic lesion in a patient with Parkinson disease may abolish rigidity while leaving the akinesia unaltered Thus it appears that apart from their contribution to the maintenance of posture, the basal ganglia provide an essential element for the performance of the large variety of voluntary and semiautomatic actions required for the full repertoire of natural human motility Hallett and Khoshbin, in an analysis of ballistic (rapid) movements in the parkinsonian patient, found that the normal triphasic sequence of agonist-antagonist-agonist activation, as described in the next chapter, is intact but lacks the energy (number of activated motor units) to complete the movement Several smaller triphasic sequences are then needed, which slows the movement The patient experiences these phenomena as not only a slowness but also a type of weakness That cells in the basal ganglia participate in the initiation of movement is also evident from the fact that the ring rates in these neurons increase before movement is detected clinically In terms of pathologic anatomy and physiology, bradykinesia may be caused by any process or drug that interrupts some component of the cortico-striato-pallido-thalamic circuit Clinical examples include reduced dopaminergic input from the substantia nigra to the striatum, as in Parkinson disease; dopamine receptor blockade by neuroleptic drugs; extensive degeneration of striatal neurons, as in striatonigral degeneration and the rigid form of Huntington chorea; and destruction of the medial pallidum, as in Wilson and Hallervorden-Spatz diseases As illustrated in Fig 4-4B, which gives a schematic representation of the hypokinetic state of Parkinson s disease, changes in the cortico-striato-pallido-thalamic circuit (in this case mainly the direct striatopallidal pathway) can be interpreted in terms of altered neurochemical and resultant physiologic connectivity within the basal ganglia The reciprocal situation, enhanced motor activity, is summarized in the analogous diagram for Huntington disease (Figure 4-4C), in which a reduction in the activity of the indirect striatopallidal pathway leads to enhanced excitatory motor drive in the thalamo-cortical motor pathway A number of other disorders of voluntary movement may also be observed in patients with diseases of the basal ganglia A persistent voluntary contraction of hand muscles, as in holding a pencil, may fail to be inhibited, so that there is interference with the next willed movement This has been termed tonic innervation, or blocking and may be brought out by asking the patient to repetitively open and close a st Attempts to perform an alternating sequence of movements may be blocked at one point, or there may be a tendency for the voluntary movement to adopt the frequency of the coexistent tremor Muscles fatigue more readily on repeated activity In still other cases, performance of a simple movement (eg, touching nger to nose, forcefully striking an object with the sted hand) is rendered impossible by the simultaneous contraction of agonist and antagonist muscles not only of the arm and hand but also of the neck, trunk, and even the legs ( intention spasm ). print barcode in excel 2010 barcode generator for excel free download - SourceForge
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